TREATMENT
PRE HOSPITAL
Rarely associated with airway emergencies, but if any signs of airway compromise are present:
- Intubation equipment at bedside
- Transport in sitting position
- Supplemental oxygen
- Suction secretions as needed
INITIAL STABILIZATION/THERAPY
- Assess for airway patency
- Establish definitive airway via endotracheal intubation or cricothyrotomy/tracheostomy in the presence of:
- Respiratory distress
- Inability to handle secretions
- Oropharyngeal tissue swelling that impairs or threatens airway
ED TREATMENT/PROCEDURES
- Analgesia with NSAIDs or opiates may be required
- Incision and drainage:
- Anesthetize gingiva superficially with 2% lidocaine with 1:100,000 epinephrine until blanching occurs
- Make a 1 cm stab incision using a scalpel blade toward alveolar bone
- Blunt dissection using mosquito hemostat
- Irrigate cavity with saline
- If abscess cavity sufficiently large, place 1/4 in iodoform gauze drain or fenestrated Penrose drain for 24–48 hr:
- To prevent its aspiration, secure gauze or drain with silk suture
- Antibiotics:
- Indicated if abscess extensive or if systemic signs present
- Penicillin considered first-line empiric therapy
- Erythromycin, azithromycin, clindamycin for penicillin-allergic patients
- Clindamycin for penicillin-allergic patients or patients not responding to penicillin
- Ampicillin/sulbactam for severe infections
- Warm salt water rinses hourly while awake for 24–48 hr
MEDICATION
First Line
- Penicillin VK: 250–500 mg PO q6h (peds: 25–50 mg/kg/d PO div. q6h)
- Azithromycin: 500 mg (peds: 10 mg/kg) PO 1st day, then 250 mg (peds: 5 mg/kg) PO per day × 4 days (for penicillin-allergic patients)
- Clindamycin: 150–450 mg PO q6h (peds: 10–25 mg/kg/d div. PO q6h)
- Clindamycin: 300–900 mg IV q8h (peds: 15–25 mg/kg/d IV div. q8h)
- Erythromycin: 250–500 mg PO q6–8h (peds: 30–50 mg/d PO div. q6h)
Second Line
- Ampicillin/sulbactam IV: 1.5–3 g IV q6h (peds >1 yr, <40 kg: 300 mg/kg/d IV div. q6h)
- Amoxicillin/clavulanate: 875 mg PO q12h (peds: 25–45 mg/kg/d div. q12h) (oral conversion)
- Moxifloxacin: 400 mg PO or IV QD (not routinely recommended for pediatric use)
FOLLOW-UP
DISPOSITION
Admission Criteria
- Severe infection or complication requiring parenteral antibiotics
- Necrosis or cellulitis involving areas with potential airway compromise
- Cavernous sinus thrombosis
- Osteomyelitis
- Outpatient therapy failure
- Immunocompromised patients:
- Neutropenia
- Uncontrolled diabetes
- Advanced HIV
- Cancer patients undergoing chemotherapy
- Ludwig angina
- Systemic involvement with significant dehydration
- Patients unable to handle secretions
- Patients unable to manage infection at home because of physical or mental disability or psychosocial factors
Discharge Criteria
- Uncomplicated cases
- Dental follow-up available in 24–48 hr
Issues for Referral
Dental follow-up useful for:
- Viability of affected tooth
- Dental extraction
- Root canal therapy
- Removal of Penrose drain or wic
FOLLOW-UP RECOMMENDATIONS
Dental follow-up in 24–48 hr:
- Lacking dental follow-up, patients should have alternative follow-up in 24–48 hr with provider familiar with disease process (oral surgeon, ED, urgent care, primary care)
PEARLS AND PITFALLS
Maxillary sinusitis may be incorrectly diagnosed without adequate oral exam:
- Dental follow-up is essential for short-term resolution of symptoms and long-term tooth viability and oral hygiene issues
ADDITIONAL READING
- Beaudreau RW. Chapter 240. Oral and dental emergencies. In: Tintinalli JE, Stapczynski JS, Cline DM, Ma OJ, Cydulka RK, Meckler GD, eds.
Tintinalli’s Emergency Medicine: A Comprehensive Study Guide
. 7th ed. New York, NY: McGraw-Hill; 2011.
- Benko K Chapter 22. Dental emergencies. In: Adams JG, ed.
Emergency Medicine
. 1st ed. Philadelphia, PA: Saunders Elsevier; 2008.
- Capps EF, Kinsella JJ, Gupta M, et al. Emergency Imaging assessment of acute nontraumatic conditions of the head and neck.
Radiographics.
2010;30:1335–1352.
- Gould J. Dental abscess. Medscape. Updated May 30, 2012.
- Levi ME, Eusterman VD. Oral infections and antibiotic therapy.
Otolaryngol Clin North Am.
2011;44:57–78.
- Patel PV, Kumar S, Patel A. Periodontal abscess: A review.
J Clin Diagn Res
. 2011;5:404–409.
- Robertson D, Smith AJ. The microbiology of the acute dental abscess.
J Med Microbiol
. 2009;58(Pt 2):155–162.
- Schaad UB. Will fluoroquinolones ever be recommended for common infections in children?
Pediatr Infect Dis J.
2007;26:865–857.
- Sobottka I, Wegscheider K, Balzer L, et al. Microbiological analysis of a prospective, randomized, double-blind trial comparing moxifloxacin and clindamycin in the treatment of odontogenic infiltrates and abscesses.
Antimicrob Agents Chemother
. 2012;56:2565–2569.
See Also (Topic, Algorithm, Electronic Media Element)
Toothache
CODES
ICD9
- 522.5 Periapical abscess without sinus
- 522.7 Periapical abscess with sinus
- 523.31 Aggressive periodontitis, localized
ICD10
- K04.6 Periapical abscess with sinus
- K04.7 Periapical abscess without sinus
- K05.21 Aggressive periodontitis, localized
PERIORBITAL AND ORBITAL CELLULITIS
Shari Schabowski
BASICS
DESCRIPTION
Periorbital Cellulitis
- An inflammatory, typically infectious condition affecting the eyelid(s)
- It is anatomically distinguished by its location, isolated to the tissues anterior to the orbital septum:
- Orbital septum is the connective tissue extension of the orbital periosteum that is reflected into the upper and lower eyelids
- Extension to the deep tissues is rare because the septum represents a nearly impenetrable barrier but it may be incomplete
- Most commonly presents as a complication of upper respiratory tract infection (URTI) and sinusitis:
- Swelling is caused by inflammatory edema from vascular and lymphatic congestion
- May occur as a complication of a localized inflammation/infection in the eyelid or adjacent structures:
- Blepharitis
- Hordeolum
- Dacryocystitis
- Surrounding skin disruptions:
- Insect bites
- Minor trauma
- Impetigo or other dermatologic disorders
Orbital Cellulitis
- Inflammatory process in the structures deep to the orbital septum
- Typically occurs secondary to extension from an adjacent structure:
- Sinusitis:
- Most commonly ethmoiditis penetrating through the thin lamina papyracea
- Dental abscess
- Retained foreign body in the orbit
- Puncture wounds
- Orbital fracture
- Postoperative infection
- Hematogenous spread from a remote source due to valveless orbital veins
- Rare cause—direct extension of periorbital cellulitis
ETIOLOGY
Periorbital Cellulitis
- Streptococcus pneumoniae
- Staphylococcus aureus
- Streptococcus pyogenes
- Moraxella catarrhalis
- Haemophilus influenzae
- Gonococcus – rare
- Consider nonbacterial cause