Rosen & Barkin's 5-Minute Emergency Medicine Consult (423 page)

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Authors: Jeffrey J. Schaider,Adam Z. Barkin,Roger M. Barkin,Philip Shayne,Richard E. Wolfe,Stephen R. Hayden,Peter Rosen

Tags: #Medical, #Emergency Medicine

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EKG:

  • Prolonged QT (most common)
  • Nonspecific ST changes
  • Premature ventricular contractions
  • Atrial fibrillation
  • Ventricular tachycardia
  • Acute MI (rare)
DIFFERENTIAL DIAGNOSIS

Other causes of coma, cardiac dysrhythmias, or trauma:

  • Hypoglycemia
  • Intoxication
  • Drug overdose
  • Cardiovascular disease
  • Cerebrovascular accident
  • Seizure
  • Syncope
TREATMENT
PRE HOSPITAL
  • Field triage should rapidly focus on providing ventilatory support to unconscious victims or those in cardiopulmonary arrest:
    • Prevents primary cardiac arrest from degenerating into hypoxia-induced secondary cardiac arrest
    • Conscious victims are at lower risk for imminent demise.
  • Spine immobilization for:
    • Cardiopulmonary arrest (suspected trauma)
    • Significant mechanical trauma
    • Suspected loss of consciousness at any time
  • Cover superficial burns with sterile saline dressings.
  • Immobilize injured extremities.
  • Rapid extrication to decrease risk for repeat lightning strikes
INITIAL STABILIZATION/THERAPY
  • ABCs
  • Standard advanced cardiac life support measures for cardiac arrest
  • Diligent primary and secondary survey for traumatic injuries and other causes of collapse/injury:
    • Maintain cervical spine precautions until cleared.
  • Treat altered mental status with glucose, naloxone, or thiamine as indicated.
  • Hypotension requires volume expansion, blood products, and/or pressor agents.
ED TREATMENT/PROCEDURES
  • IV access
  • Cardiac monitor and pulse oximetry
  • Clean and dress burns.
  • Tetanus prophylaxis
  • Treat myoglobinuria if present:
    • Diuretics, such as furosemide or mannitol
    • Alkalinize urine to a pH of 7.45 with IV sodium bicarbonate
  • Volume expansion:
    • Do not follow burn treatment formulas because lightning burns are rarely the cause of fluid loss.
    • Occult deep burn injury is rare when compared with other types of electrical current injury.
    • Titrate volume administration to urine output.
    • Fluid loading may be dangerous if patient has concomitant head injury.
  • Compartment syndrome:
    • Must be distinguished from vasospasm, autonomic dysfunction, and paralysis, which are usually self-limited phenomena.
    • Fasciotomy will rarely be necessary.
  • NSAIDs and high-dose steroids have been proposed to reduce long-term neurologic and corneal damage.
MEDICATION
  • Furosemide: 1 mg/kg IV slow bolus q6h
  • Mannitol: 0.5 mg/kg IV, repeat PRN
  • Sodium bicarbonate: 1 amp IV push (peds: 1 mEq/kg) followed by 2–3 amps/L D5W IV fluid
FOLLOW-UP
DISPOSITION
Admission Criteria
  • Postcardiac arrest patients
  • History of change in mental status/altered level of consciousness
  • History of chest pain, dysrhythmias, or ECG changes:
    • May not resolve spontaneously
    • 24–48 hr observation period to identify potentially unstable cases
    • Myoglobinuria
    • Acidosis
    • Extremity injury with or at risk for compartment syndrome
Discharge Criteria

Asymptomatic patients with no injuries

FOLLOW-UP RECOMMENDATIONS
  • Close follow-up with subspecialists may be required due to the risk for delayed sequelae:
    • Neurology:
      • Memory deficit
      • Attention deficit
      • Aphasia
      • Sleep disturbance
      • Prolonged paresthesia and dysesthesias
    • Ophthalmology
    • ENT
  • Psychology/psychiatry:
    • Anxiety
    • Depression
    • Personality changes
    • Post-traumatic stress disorder
PEARLS AND PITFALLS
  • Do not follow burn treatment formulas for lightning burns and injuries.
  • Be diligent in the primary and secondary survey so as not to miss occult injuries.
  • Have a low threshold to admit and monitor patients with cardiopulmonary complaints, as unstable dysrhythmias may occur 24–48 hr post injury.
ADDITIONAL READING
  • Cooper MA, Andrews CJ, Holle RL. Lightning injuries. In: Auerbach PS, ed.
    Wilderness Medicine
    . 5th ed. St. Louis, MO: Mosby; 2007:67–108.
  • Cooper MA, Holle RL. Mechanisms of lightning injury should affect lightning safety messages. 21st International Lightning Detection Conference. April 19–20, 2010; Orlando, FL.
  • O’Keefe Gatewood M, Zane RD. Lightning injuries.
    Emerg Med Clin North Am
    . 2004;22(2):369–403.
  • Price T, Cooper MA. Electrical and lightning injuries. In: Marx JA, Hockenberger RS, Walls RM, et al., eds.
    Rosen’s Emergency Medicine
    . 6th ed. Philadelphia, PA: Mosby; 2006.
See Also (Topic, Algorithm, Electronic Media Element)

Electrical Injury

CODES
ICD9
  • 949.0 Burn of unspecified site, unspecified degree
  • 994.0 Effects of lightning
  • 994.8 Electrocution and nonfatal effects of electric current
ICD10
  • T30.0 Burn of unspecified body region, unspecified degree
  • T75.00XA Unspecified effects of lightning, initial encounter
  • T75.09XA Other effects of lightning, initial encounter
LITHIUM POISONING
Sean M. Bryant
BASICS
DESCRIPTION
  • GI absorption is rapid:
    • Regular release: Peak serum levels 2–4 hr
    • Sustained release: Peak serum levels 4–12 hr
  • Half-life 24 hr
  • Slow distribution (at least 6 hr)
  • Volume of distribution 0.6–0.9 L/kg
  • Elimination:
    • Not
      metabolized
    • Renal excretion (unchanged)
    • Reabsorbed in the
      proximal
      tubules by sodium transport mechanism
    • Elimination half-life (therapeutic) is 20–24 hr and prolonged in chronic users
  • Therapeutic and toxic indices:
    • Therapeutic and toxic effects occur
      only
      when lithium is intracellular
    • Narrow toxic-to-therapeutic ratio
    • Therapeutic level 0.6–1.2 mEq/L (postdistribution)
    • Because of small size, renal handling is similar to sodium, potassium, and magnesium
  • Risk factors:
    • Acute conditions increasing risk of toxicity:
      • Dehydration (larger percent reabsorbed)
      • Overdose
    • Chronic conditions:
      • Hypertension
      • Diabetes mellitus
      • Renal failure
      • Congestive heart failure
      • Advanced age
      • Dose change
      • Drug interactions
      • Lithium therapy
      • Low-salt diet
    • The following may result in increased serum lithium levels due to decreased renal clearance or exacerbated effects:
      • NSAIDs
      • Thiazide diuretics
      • ACE inhibitors
      • Phenytoin
      • Tricyclic antidepressants
      • Phenothiazines
ETIOLOGY
  • Acute or chronic conditions affecting lithium clearance
  • Overdose
DIAGNOSIS

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